Pending ReviewPCAC July 23, 2026

TB-500

Thymosin Beta-4 Synthetic Analog

Half-life
Not established
Route
SC / IM
Loading dose
2–2.5 mg 2×/wk
Reconstitutable
Yes

What is TB-500?

TB-500 is a synthetic analog of Thymosin Beta-4 (Tβ4), a 43-amino-acid protein expressed in virtually all human and animal cells. Tβ4 is a critical regulator of actin polymerization — the process by which cells form their internal scaffolding and enable movement, division, and repair.

The key functional region of Thymosin Beta-4 is a short actin-binding segment (LKKTETQ), and TB-500 is specifically designed around this domain. This makes TB-500 a targeted version of the full protein, with the portion believed responsible for the majority of its tissue repair effects.

TB-500 promotes cell migration, differentiation, and angiogenesis — making it useful in preclinical research on wound healing, muscle repair, cardiac tissue regeneration, and inflammation reduction. Unlike BPC-157, which is gut-derived, TB-500 is a skeletal and cardiac repair-focused compound.

Research Evidence

SilverWound & Skin Healing

Tβ4 (the parent compound) has completed Phase II clinical trials showing accelerated healing of diabetic foot ulcers and pressure ulcers. TB-500 as a synthetic analog extrapolates these findings; direct human trials for TB-500 specifically are limited.

SilverCardiac Repair

Strong preclinical data in rodent models showing Tβ4 promotes cardiac progenitor cell activation and reduces infarct size post-MI. Thymosin Beta-4 has entered clinical cardiac trials separately.

BronzeMuscle & Connective Tissue

Animal models show improved healing of muscle tears and ligament injuries. Mechanism consistent with actin regulation and VEGF upregulation.

BronzeInflammation Reduction

Tβ4 modulates NF-κB signaling, reducing inflammatory cytokine production in several preclinical models. Effect in the synthetic TB-500 analog is assumed but not directly confirmed in all models.

Evidence grades: Gold = RCT human data · Silver = multiple animal studies or parent compound human data · Bronze = limited or preliminary

Dosing Protocols

Loading phase
2–2.5 mg 2× per week
4–6 weeks. Inject SC or IM. Site rotation recommended.
Maintenance phase
2–2.5 mg every 2 weeks
After loading. Used for ongoing recovery support.
Acute injury protocol
2 mg 3× per week
More aggressive loading for acute muscle or tendon injuries. Run for 4–8 weeks.
Stack with BPC-157
Often combined
The "Wolverine Stack" combines BPC-157 (250–500 mcg/day) + TB-500 (2 mg 2×/wk) for synergistic tissue repair.

Reconstitution

TB-500 is supplied as a lyophilized powder. Reconstitute with bacteriostatic water. Common vial sizes are 2 mg and 5 mg.

VialBAC WaterConcentration2 mg draw
2 mg1 ml2,000 mcg/ml100 units (1.0 ml)
5 mg2.5 ml2,000 mcg/ml100 units (1.0 ml)
5 mg5 ml1,000 mcg/ml200 units (2.0 ml)
Calculate exact protocol →

Frequently Asked Questions

What is TB-500?

TB-500 is a synthetic analog of Thymosin Beta-4, a naturally occurring protein involved in actin regulation, tissue repair, and cell migration. It is researched for wound healing, injury recovery, inflammation reduction, and cardiac repair.

What is the TB-500 dosing protocol?

Common protocols use a loading phase of 2–2.5 mg twice weekly for 4–6 weeks, followed by maintenance of 2–2.5 mg every 2 weeks. Some combine TB-500 with BPC-157 for synergistic injury repair.

Is TB-500 legal in 2026?

TB-500 is currently FDA Category 2, meaning it cannot be compounded by licensed 503A pharmacies. The FDA PCAC is reviewing TB-500 on July 23, 2026 for potential Category 1 reclassification.

How does TB-500 differ from BPC-157?

BPC-157 is a gut-derived peptide with strong GI and tendon healing effects. TB-500 is derived from Thymosin Beta-4 and works primarily through actin regulation and cardiac/skeletal muscle repair. They are often stacked together for comprehensive injury recovery (the Wolverine Stack).

Disclaimer: For informational and research purposes only. Not medical advice.

This profile was prepared using AI-assisted research synthesis. Citations are provided where applicable — verify with primary sources before clinical application.